The process of forgetting was first studied scientifically by Hermann Ebbinghaus, a German experimental psychologist, who performed memory tests with groups of nonsense syllables (disconnected syllables without associative connection). Ebbinghaus showed that the rate of forgetting is greatest at first, gradually diminishing until a relatively constant level of retained information is reached. Theories to explain forgetting include the concept of disuse, which proposes that forgetting occurs because stored information is not used, and that of interference, which suggests that old information interferes with information learned later and new information interferes with previously learned information.
In some instances, memory loss is an organic, physiological process. Retrograde amnesia , i.e., the failure to remember events preceding a head injury, is evidence of interrupted consolidation of memory. In anterograde amnesia, events occurring after brain damage—e.g., in head injury or alcoholism—may be forgotten. Memory loss may also result from brain cell deterioration following a series of strokes, cardiovascular disease, or Alzheimer's disease (see dementia ).
Physiologically, learning involves modification of neural pathways. PET scans and related studies have shown certain parts of the brain, such as the frontal lobe of the cerebral cortex and a structure called the hippocampus, to be particularly active in recall. Computer models of brain memory are called neural networks . In a study using genetic manipulation, a mouse with enhanced memory capabilities has been produced.
See M. H. Ashcroft, Human Memory and Cognition (1989, repr. 1994); N. Cowan, Attention and Memory (1995, repr. 1998); J. McConkey, ed. The Anatomy of Memory (1996); D. L. Schacter, Searching for Memory (1996) and The Seven Sins of Memory (2001); J. A. Groegerd, Memory and Remembering (1997); A. Baddeley, Human Memory (rev. ed. 1998); R. Rupp, Committed to Memory (1998).
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